Patterns of gene expression during epileptogenesis: Microarray studies in rats

J. A. Gorter, F. H. Lopes da Silva, E. Aronica

Research output: Chapter in Book/Report/Conference proceedingChapterAcademicpeer-review

Abstract

Microarray experiments with RNA from brain regions obtained at different time points after electrically- or pharmacologically-induced status epilepticus in the rat, point to inflammation as a possible contributing epileptogenic factor. The overwhelming acute inflammatory response is reflected in a general and time dependent activation of multiple genes that code proteases, cytokines and other inflammatory substances. Expression of these genes can alter the dynamics of neuron-glia networks-for example, by potentiating glutamate release from neuronal and glial sources. Some over-activated genes may not only cause parenchymal inflammation but also inflammation of endothelial cells, leading to disruption of the blood brain barrier-a feature that may further contribute to network instability. From this scenario it can be speculated that anti-inflammatory treatment could help to slow down the process of epileptogenesis or prevent further progression of the disease.
Original languageEnglish
Title of host publicationThe Curated Reference Collection in Neuroscience and Biobehavioral Psychology
PublisherElsevier Science Ltd
Pages240-250
ISBN (Electronic)9780128093245
DOIs
Publication statusPublished - 2016

Publication series

NameThe Curated Reference Collection in Neuroscience and Biobehavioral Psychology

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