Abstract
In this issue of Blood, Moxon et al provide novel insight into the pathogenesis of cerebral malaria, linking loss of the endothelial protein C receptor (EPCR) on brain vessels, caused by cytoadherent infected erythrocytes, with localized coagulation, inflammation, and disruption of endothelial barrier function
Original language | English |
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Pages (from-to) | 624-625 |
Journal | Blood |
Volume | 122 |
Issue number | 5 |
DOIs | |
Publication status | Published - 2013 |