Therapeutic Strategies for Targeting CDKN2A Loss in Melanoma

Inger Z. M. Kreuger, Roderick C. Slieker, Tim van Groningen, Remco van Doorn

Research output: Contribution to journalReview articleAcademicpeer-review

15 Citations (Scopus)

Abstract

Loss of the tumor suppressor gene CDKN2A, encoding p16 and p14, is a frequent event driving melanoma progression. Therefore, therapeutic strategies aimed at CDKN2A loss hold great potential to improve melanoma treatment. Pharmacological inhibition of the p16 targets CDK4/6 is a prime example of such a strategy. Other approaches exploit cell cycle deregulation, target metabolic rewiring, epigenetically restore expression, act on dependencies resulting from co-deleted genes, or are directed at the effects of CDKN2A loss on immune responses. This review explores these therapeutic strategies targeting CDKN2A loss, which potentially open up new avenues for precision medicine in melanoma.
Original languageEnglish
Pages (from-to)18-25.e1
JournalJournal of Investigative Dermatology
Volume143
Issue number1
Early online date2022
DOIs
Publication statusPublished - Jan 2023

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