Transient hypothyroxinemia in juvenile glycoprotein hormone subunit B5 knock-out mice

Clementine J. J. van Zeijl, Olga V. Surovtseva, Wilmar M. Wiersinga, Anita Boelen, Eric Fliers

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18 Citations (Scopus)

Abstract

The heterodimer thyrostimulin, comprised of two novel glycoprotein hormone subunits GPA2 and GPB5, activates the TSH receptor. To understand its role in the regulation of the hypothalamus-pituitary-thyroid (HPT-) axis, we evaluated juvenile and adult GPB5 knock-out (GPB5(-/-)) and wild type mice own during euthyroidism, hypothyroidism and thyrotoxicosis Surprisingly, juvenile euthyroid GPB5(-/-) mice displayed marked hypothyroxinemia (25% lower serum T-4, unchanged TSH) and also during thyrotoxicosis juvenile GPB5(-/-) mice had 25% lower serum T-4, compared to WT During hypothyroidism, despite similar serum T-4, pituitary TSH beta mRNA was 2-fold lower in GPB5(-/-) mice compared to WT Adult mice displayed increased pituitary deiodinase type 2 during euthyroidism and decreased serum T-4 during hypothyroidism in GPB5(-/-). Thus, lacking GPB5 results in moderate deviations of the HPT-axis. The more pronounced differences observed in juvenile mice compared to adult mice are in agreement with the notion that GPB5 has a role during development (C) 2010 Elsevier Ireland Ltd All rights reserved
Original languageEnglish
Pages (from-to)231-238
JournalMolecular and cellular endocrinology
Volume321
Issue number2
DOIs
Publication statusPublished - 2010

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